MagezGroup

Antibody-mediated control of Trypanosoma vivax infection fails in the absence of tumour necrosis factor.

La Greca, F., C. Haynes, B. Stijlemans, C. De Trez, and S. Magez, "Antibody-mediated control of Trypanosoma vivax infection fails in the absence of tumour necrosis factor.", Parasite Immunol, vol. 36, issue 6, pp. 271-6, 2014 Jun.

Trypanosomiasis-induced B cell apoptosis results in loss of protective anti-parasite antibody responses and abolishment of vaccine-induced memory responses.

Radwanska, M., P. Guirnalda, C. De Trez, B. Ryffel, S. Black, and S. Magez, "Trypanosomiasis-induced B cell apoptosis results in loss of protective anti-parasite antibody responses and abolishment of vaccine-induced memory responses.", PLoS Pathog, vol. 4, issue 5, pp. e1000078, 2008 May.

The role of B-cells and IgM antibodies in parasitemia, anemia, and VSG switching in Trypanosoma brucei-infected mice.

Magez, S., A. Schwegmann, R. Atkinson, F. Claes, M. Drennan, P. De Baetselier, and F. Brombacher, "The role of B-cells and IgM antibodies in parasitemia, anemia, and VSG switching in Trypanosoma brucei-infected mice.", PLoS Pathog, vol. 4, issue 8, pp. e1000122, 2008.

iNOS-producing inflammatory dendritic cells constitute the major infected cell type during the chronic Leishmania major infection phase of C57BL/6 resistant mice.

De Trez, C., S. Magez, S. Akira, B. Ryffel, Y. Carlier, and E. Muraille, "iNOS-producing inflammatory dendritic cells constitute the major infected cell type during the chronic Leishmania major infection phase of C57BL/6 resistant mice.", PLoS Pathog, vol. 5, issue 6, pp. e1000494, 2009 Jun.

T. brucei infection reduces B lymphopoiesis in bone marrow and truncates compensatory splenic lymphopoiesis through transitional B-cell apoptosis.

Bockstal, V., P. Guirnalda, G. Caljon, R. Goenka, J. C. Telfer, D. Frenkel, M. Radwanska, S. Magez, and S. J. Black, "T. brucei infection reduces B lymphopoiesis in bone marrow and truncates compensatory splenic lymphopoiesis through transitional B-cell apoptosis.", PLoS Pathog, vol. 7, issue 6, pp. e1002089, 2011 Jun.

Adenylate cyclases of Trypanosoma brucei inhibit the innate immune response of the host.

Salmon, D., G. Vanwalleghem, Y. Morias, J. Denoeud, C. Krumbholz, F. Lhommé, S. Bachmaier, M. Kador, J. Gossmann, F. Braga Steh Dias, et al., "Adenylate cyclases of Trypanosoma brucei inhibit the innate immune response of the host.", Science, vol. 337, issue 6093, pp. 463-6, 2012 Jul 27.

Tsetse salivary gland proteins 1 and 2 are high affinity nucleic acid binding proteins with residual nuclease activity.

Caljon, G., K. De Ridder, B. Stijlemans, M. Coosemans, S. Magez, P. De Baetselier, and J. Van Den Abbeele, "Tsetse salivary gland proteins 1 and 2 are high affinity nucleic acid binding proteins with residual nuclease activity.", PLoS One, vol. 7, issue 10, pp. e47233, 2012.

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